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Prior genetic and postmortem studies have revealed abnormalities in these neurotransmission systems in ASD: genetic studies have revealed altered single-nucleotide polymorphisms (SNPs) in genes related to Glu transporters [6], and postmortem studies have demonstrated the overexpression of mRNA encoding Glu receptors [7]

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Concerning GSTIs, according to the existence of clearly visible N-terminal extensions in some representative members, several prediction programs indicate that they could be targeted either to the chloroplasts or to mitochondria, although this remains to be demonstrated experimentally